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Fig. 5. Mean c.f.u. counts in organs of untreated and drug-treated beige mice infected with ST M. celatum. Standard deviations 20 % ; are not shown for clarity. Mice five per group per time point ; were inoculated i.p. with 107 c.f.u. and treatments were begun 1 d later. #, CTL ; , INH ; , CLA ; $, AZI ; 7, EMB ; , RMP ; 4, RFB ; 5, CIP!
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HGF administration abrogated colitis-associated weight loss in rats with DSS-induced colitis To examine the effect of intraperitoneally-administered HGF on the severity of DSS-induced colitis, the body-weights of HGF or mock-treated rats were measured Fig. 2 ; . Initially, sixty-five rats were treated with 5% DSS, and were divided into PBS- or HGF-treated groups, consisting of 35 or animals, respectively Table 1 ; . Seven rats from each group died within 2 days after osmotic pump implantation on days 6 and 7 ; , but all rats survived the subsequent 1% DSS administration from days 8 to11 ; . The body-weights of rats with DSS-induced colitis gradually increased during the five days of 5% DSS administration, and liquid stool and gross bleeding from the rectum were induced by this treatment. Intraperitoneal administration of PBS or recombinant human HGF was started on day 5. Although gross bleeding disappeared regardless of HGF-treatment during 4 days of 1% DSS administration, persistent liquid stool and wasting were observed in rats treated with PBS, resulting in severe weight loss. Rats treated with recombinant human HGF, however, suffered less severe diarrhea and weight loss. The body-weights of HGF-treated rats on days 9 and 11 248 17.9 g and 259.0 26.0 g, respectively ; were significantly higher than those of PBS-treated rats 238.1 31.0 g and 235.7 24.4 g, respectively ; p 0.033 and p 0.012, respectively ; . These results indicate that the administration of recombinant human HGF significantly abrogated colitis associated weight loss in rats with DSS-induced colitis.
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After the initial 6 weeks is known to be the strongest prognostic variable for outcome. In Section II, Dr. Yasodha Natkunam reviews the features of SHML, which most often presents as painless cervical lymphadenopathy, although many patients can have extranodal involvement as well. These sites include the skin, respiratory tract, bone, lung, gastrointestinal tract, and brain. The diagnosis rests on finding intact lymphocytes in the cytoplasm of activated macrophages as well as accumulation of mature plasma cells. Hemolytic or non-hemolytic anemias, hypergammaglobulinemia, and elevated erythrocyte sedimentatin rate ESR ; are often found with SHML. An intriguing finding of human herpesvirus HHV ; -6 viral proteins in SHML has been reported in several patients, but needs further study. SHML associated with lymphoproliferations triggered by defects in apoptosis are discussed since this mechanism may provide a clue to the etiology. Therapy for SHML varies greatly in reported case series. Many patients have spontaneous regression or resolution after surgical removal of isolated node groups. Others with systemic involvement may benefit from chemotherapy, but no clinical trials have been done. In Section III, Dr. Steven Swerdlow clarifies key features of the four types of CD. Localized cases are divided into the hyaline vascular type and plasma cell type. Both are usually cured by surgical excision and have symptoms mainly of a mass lesion, although the latter often also has constitutional symptoms. The two types are distinguished largely by the nature of the follicles and the number of interfollicular plasma cells. Interleukin IL ; -6 expression is increased in the plasma cell type. Multicentric CD of the plasmablastic type is most often found in HIVpositive patients with coincident HHV-8 infection. Many have lymphomas or Kaposi sarcomas. Other cases of multicentric CD are also most like the plama cell type, however, with disseminated disease and constitutional symptoms. A wide variety of anti-neoplastic drugs, radiation therapy, anti-IL-6 and rituximab or atlizumab have been used with varying success in patients with.
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